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The addition of hyperhomocysteinemia to Doppler alterations increased the sensitivity from 66.7 to 77.8%.
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These alterations increase the difficulty of joint replacement, and may also threaten the long-term survival of the prosthetic implant.
None of the modified genes has a direct link to cancer, however, making it unclear whether these chemical alterations increase the risk of developing the disease.
These alterations increase the sensitivity of detection [ 17].
Sleep alterations increase the pathological significance of any disease and reduce general well-being [ 7- 10].
Dietz [ 9] defined it as "a developmental stage in which physiologic alterations increase the later prevalence of obesity".
Furthermore, sleep alterations increase the pathologic significance of any disease and reduce general well-being [ 16, 17].
During tumour expansion there is a constant acquisition of genetic and epigenetic alterations, increasing the intra-tumor heterogeneity, and making difficult the development of effective therapies [ 2].
These neurophysiological alterations increase the efficacy of usage of relevant brain networks and enable the brain to compensate for disrupted networks (this is captured in the term 'brain reserve').
Although genetic alterations increase the production of A β in rare familial AD, reduced A β clearance from the brain likely accounts for sporadic AD, which is much more common.
There is ongoing debate whether pathogenic RAD51C alterations increase the relative risk for BC in addition to that for OC, which was estimated to be 5.88 (95% confidence interval = 2.91 to 11.88; P = 7.65 × 10 7) [ 2].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com