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Noncancerous idiopathic lesions (e.g., foci of cellular alteration) were found primarily in fish from the DR, compared with the reference sites (Malins et al. 1987).
Novel regions of copy number alteration were found to be common to LCIS, including gain at 20q13.13 and loss at 19q13.2-q13.31.
At least two alterations of the VHL gene, potentially leading to biallelic alteration, were found in 68 tumours (66.7%), whereas no or a single alteration of the VHL gene was found in 34 patients (33.3%).
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No alteration was found in the number of crossings and rearings of animals treated with melatonin and/or quinolinic acid.
However, no obvious alteration was found in p-NMDAR1/NMDAR1 ratio.
This alteration was found exclusively in 4 EMB-resistant isolates.
The c.677C > T alteration was found to be an important risk factor for cerebral events.
No signs of inflammation or any other alteration was found in the head kidney tissue.
In this study, no copy number alteration was found for BCL2 and BCL-XL gene.
However, this alteration was found to be reversibly associated with the proliferative rate of individual foci (Neveu et al. 1994).
The same study was conducted by Fattahi and his colleagues in which no Ashkenazi Jews alteration was found [ 137].
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