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No obvious alteration was apparent in the morphology of Golgi tendon organs and Pacinian corpuscles in co-IgNrg1 mutants, nor did we detect changes in myelination of peripheral nerves (Supplementary Figure S5B and C, g-ratios P12: 0.72 ±0.02 and 0.71 ±0.01 in the sciatic nerve of control and co-IgNrg1 mice, respectively).
Similar(57)
This pattern were changed 24 h post-SE, when histopathological alterations were apparent in both the superficial (Fig. 5and) and inferior (Fig. 5B″') layers.
Similarly, at the metabolite level, only minor alterations were apparent in response to hypoxia (in contrast to WT).
Two alterations were apparent in melanoma apoptosis pathway: elevation of caspase 8 transcription and an E2f1-to- E2f4 gene isoform switch.
Such alterations are apparent in the early stages and continue to escalate in both receptor types with disease progression, emphasizing the importance of this growth regulatory pathway in gastric carcinogenesis.
DCC alterations are apparent in the early stages, and UNC5C alterations escalate with the progression of the disease, suggesting that the cumulative alterations of netrin-1 receptors was a late event in gastric cancer progression and emphasizing the importance of this growth regulatory pathway in gastric carcinogenesis.
No phenotypic change or alteration in ESC markers was apparent in cells maintained in activin plus Fgf.
This morphological alteration was not apparent after single-agent treatment.
Although a clear trend was apparent for alterations in SV by recovery condition, this did not reach significance (p = 0.06).
In further support of this finding, no alteration in the abundance of astrocytes was apparent in mixed cultures following A β treatment (Supplementary Figure 1).
It was apparent that K209 had a genomic alteration in the molecular mechanism responsible for Pb1-mediated resistance gene activation.
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