The phrase "alteration of receptor" is correct and usable in written English.
It can be used in contexts related to biology, pharmacology, or medical discussions, particularly when discussing changes to receptor structures or functions.
Example: "The alteration of receptor sensitivity can significantly impact drug efficacy and patient outcomes."
Alternatives: "modification of receptor" or "change in receptor".
Though the variant 1911G was associated with elevated protein levels in a human kidney cell line expression system [23], the investigation of functional responses to capsaicin, pH and temperature in whole-cell patch-clamp and calcium imaging experiments suggests no critical alteration of receptor structure and function with respect to this receptor variant [24].
Arboviruses adapt readily to new susceptible hosts by alteration of receptor specificity, transmission efficiency, antigenicity, and ecological and environmental conditions.
This leads to a range of biochemical and immunological changes, including reduction of prostaglandin E2 synthesis, alteration of receptor expression and activity, and reduced pro-inflammatory cytokine responses.
In addition, these possible conformational changes might be responsible for a lower receptor affinity that determines an alteration of receptor bond and therefore of the correlated signalling of response.
Gene copy number variation (CNV) is responsible for several important phenotypes of the malaria parasite Plasmodium falciparum, including drug resistance, loss of infected erythrocyte cytoadherence and alteration of receptor usage for erythrocyte invasion.
The results further opened the opportunities to develop the tailored sensory materials through the appropriate alteration of receptors in the side chain and the conjugated backbone.
This clinical instability is reflected in our present cell line model, again underlining the suitability of LTED studies for investigating the time related alteration of receptors during conditions which mimic endocrine therapy with aromatase inhibition.
Alterations of receptor tyrosine kinases (RTKs) are especially prevalent in GBM.
Thus, it seems that male reproductive function may be affected by AhR-inducing agents, but it is likely that the most sensitive period to reproductive disturbances is the fetal period where small alterations of receptor activity may be of crucial importance for development of reproductive organs.
Thus, it is possible that alterations of receptor-mediated signaling pathways could contribute to protection or exacerbation of cell death cascades in the symptomatic and/or presymptomatic phases of HD [ 67].
This high-affinity binding leads to a conformational alteration of the receptor, nuclear translocation and homodimerisation of the receptor complex.
