The phrase "alteration may contribute" is correct and usable in written English.
It can be used in contexts discussing changes or modifications that might have an impact on a situation or outcome.
Example: "In the study, we found that alteration may contribute to improved performance in the experimental group."
Alternatives: "modification might aid" or "change could enhance".
This alteration may contribute to incorrect identification of a vertebral segment, leading to wrong-level spine surgery and poor correlation with clinical symptoms.
Such "wiring" alteration may contribute not only to the behavioral deficits associated with this pharmacological model, but also to the disease pathogenesis.
This alteration may contribute to increasing neuronal excitability in the BNST and anxiety in the stressed rats.
Therefore, it is possible that epigenetic alteration may contribute to the increase of LRP6 protein content/phosphorylation by HNE.
Identifying relevant genetic variants or single nucleotide polymorphisms (SNPs) in genes intimately linked to various neurobiological pathways, whose alteration may contribute to suicidal behaviour, may provide insight into the etiological processes that underlie suicidal behaviour and identify novel biological therapeutic targets.
Further, considering the inflammation associated function of such gene, such mutations and expression alteration may contribute to the process of tumorigenesis through two individual regulation mechanisms: proliferation associated pathways that involve NF-kappa-B NF-kappa-B NF-kappa-Besponse andociated pathwayspecificor mimmuneviresponse[ 46].
In conjunction with altered mitochondrial function and motility, tubulin cytoskeleton alterations may contribute to the accumulation of protein aggregates and autophagic vesicles, and/or associated substrates, as observed in our sPD cybrids model.
TOP2A alterations may contribute to pathogenesis in HER2+ breast carcinomas by altering sensitivity to anthracyclines [ 31].
In these tumours with an altered control of genomic stability, accumulation of chemotherapy-induced genetic alterations may contribute to cell death and account for complete response.
These structural and functional alterations may contribute to the cognitive deficits seen in infants at risk for CH.
It has been suggested that sleep alterations may contribute to impaired QOL in BD possibly due to the effects of poor sleep quality on concentration, cognition, and memory (Giglio et al. 2008).
