The phrase "alter the release of" is correct and usable in written English.
It can be used when discussing changes to the timing or conditions under which something is made available or distributed.
Example: "The company decided to alter the release of the new product to better align with market demand."
Alternatives: "change the launch of" or "modify the distribution of".
Cannabinoids are a family of chemical compounds that bind to cannabinoid receptors found on various cell types in the body, and alter the release of neurotransmitters.
Various Toll-like receptors (TLRs) expressed in EC cells recognize different pathogen-associated molecular patterns and alter the release of polypeptide hormones and other bioactive molecules.
It has accounted to alter the release of neurotransmitters by affecting cyclic adenosine monophosphate (cAMP) concentrations which blocks inositol metabolism that results in depletion of cellular inositol and inhibition of phospholipase C-mediated signal transduction (Hardman et al. 2001).
However, noxious mechanical stimuli do not alter the release of MELM from neural segments related to the stimulated area of the body, but increase its release from other segments [2].
Secondly, the pharyngeal muscle is more sensitive to ACh than the body wall muscle, or finally low levels RHO-1(G14V) may alter the release of something other than ACh.
For example, a noxious mechanical stimulus does not alter the release of Met-enkephalin-like material (MELM) from segments of the spinal cord related to the stimulated area of the body, but does increase its release from other segments.
The interaction may take place at presynaptic terminals, altering the release of neurotransmitter, or it may take place postsynaptically, altering the effect of neurotransmitter.
The sensory of PAMPs on pathogens by PRRs on DCs is passed to T and/or B cells through altering the release of cytokines, the expression of co-stimulatory molecules and the up-regulation of integrins for adhesion.
Importantly, neither stimulation with TNF-α and IFN-γ for 48 h nor exposure to 1% O2 for 24 h followed by a reoxygenation period of 24 h altered the release of sJAM-A as detected by Western blotting which confirmed the results obtained by ELISA (Figure 4F).
In addition, the same signalling pathway in microglial cells seems to modulate the progress of demyelination by altering the release of proinflammatory components such as TNFα and iNOS.
Interventions that altered the release of anti-inflammatory cytokines (IL-10, IL-1RA), antigen-presenting capacity, or bactericidal capacity were considered modulators of CARS.
