The phrase "alter the host" is correct and usable in written English.
It can be used in contexts related to technology, biology, or any situation where a change or modification to a host entity is being discussed.
Example: "In order to improve the system's performance, we need to alter the host configuration."
Alternatives: "modify the host" or "change the host."
Previous experimental studies have supported the first possibility, showing that alterations in the human gastrointestinal tract microbiota can alter the host immune response and that intestinal bacterial overgrowth, even in the absence of tissue injury or gut ischemia, can impair systemic immune responsiveness [8], [9].
Previous experimental studies have shown that alterations in the flora of the gastrointestinal tract can alter the host immune response and that intestinal bacterial overgrowth, even in the absence of tissue injury or gut ischemia, can impair systemic immune responsiveness [8], [9].
This first report of a single mutation that was sufficient to alter the host tropism of a microorganism during its evolution highlights the capacity of some pathogens to readily expand into new host species populations.
Exotoxins are a group of soluble proteins that are secreted by the bacterium, enter host cells, and catalyze the covalent modification of a host cell component(s) to alter the host cell physiology.
Biologic surgical mesh materials composed of mammalian extracellular matrix (ECM) have been successfully used to alter the host default response to injury, and induce functional, site-appropriate tissue regeneration.
Not surprisingly, pathogens can also alter the host miRNA profile to their own benefit, which is of major importance to scientists addressing high morbidity and mortality infectious diseases such as tuberculosis.
Here we use a modified version of Sasaki's multilocus gene-for-gene model [5] to explore how different combinations of host and parasite epistasis alter the host-parasite coevolutionary dynamics.
In addition to altering the host genome, these methods can target pathogens to engineer molecular immunity.
Plug-in, also called add-on or extension, computer software that adds new functions to a host program without altering the host program itself.
miR-17 sequestration by pestiviruses conferred reduced AGO binding and functional de-repression of cellular miR-17 targets, thereby altering the host transcriptome.
It is well-known that cystatins play an important role in the regulation of a whole range of physiological processes in parasites (including their digestion), and it is possible that they also have a key function in altering the host's immunity34.
