The phrase "alter synaptic plasticity" is correct and usable in written English.
It can be used in contexts related to neuroscience, psychology, or discussions about brain function and learning.
Example: "Research has shown that certain experiences can alter synaptic plasticity, leading to improved memory retention."
Alternatives: "modify synaptic plasticity" or "change synaptic plasticity."
Drugs of abuse, including METH, can cause long-lasting changes in neuronal systems [27], [28], and alter synaptic plasticity [29], [30].
These results indicate that the acute disruption of xCT in WT mice or supplementation of extracellular glutamate in sut mice did not alter synaptic plasticity.
Given the important role that the BNST plays in regulating negative affective states and negative reinforcement, several studies have examined the ability of chronic ethanol exposure to alter synaptic plasticity in this brain region.
animal experiments have shown that chemicals of the type used in the present study inhibit cognitive function (MacLusky et al. 2005), alter synaptic plasticity (Kawato 2004), and affect the acquisition of memory (Carr et al. 2003).
Exposure to drugs of abuse, in particular ethanol, is a life experience that can activate or alter synaptic plasticity, often resulting in increased drug seeking and taking and in many cases addiction.
Environmental toxicants that have a potential for altering RyR function can influence neuronal excitability, alter synaptic plasticity, and activate cytosolic and nuclear transcriptional events implicated in activity-dependent dendritic growth (Pessah et al. 2010).
Several mechanisms may account for the altered synaptic plasticity observed in neurabin KO mice.
Thus, the transcriptome data suggest that A53T-SNCA overexpression leads to a progressively diminished postsynaptic DA response and altered synaptic plasticity in the striatum.
Assessing the suggestion of altered synaptic plasticity in the transcriptome profile on a functional level, we studied LTD in corticostriatal slices from PrPmtA, PrPmtB and WT mice.
However, other major markers of aging, including increased inflammatory signaling (based on cytokine array measures in the periphery and microarray measures in the hippocampus), impaired cognition, and altered synaptic plasticity, were not altered with PIO treatment.
Next, we tested whether Salm3−/− synapses display altered synaptic plasticity.
