The phrase "alter insulin secretion" is correct and usable in written English.
It can be used in contexts related to biology, medicine, or health, particularly when discussing how certain factors or substances can change the amount of insulin produced by the pancreas.
Example: "Certain medications can alter insulin secretion, impacting blood sugar levels in diabetic patients."
Alternatives: "modify insulin release" or "change insulin production".
In this observational study performed in two patients with PHHI, the fate of orally supplemented dl sodium β-OHB has been documented by increased concentrations of β-OHB in blood, CSF, and brain, as shown by cerebral in vivo proton MRS. At least in our two patients, dl sodium β-OHB did not seem to alter insulin secretion and was tolerated without side effects.
Medical records were reviewed for the presence or absence of diabetes, complications of diabetes, treatment of diabetes or concomitant medications known to directly alter insulin secretion or sensitivity.
PSN632408 does not alter insulin secretion in the presence of 2.8 m M glucose.
Variation in GLP1R may alter insulin secretion in response to exogenous GLP-1.
The possibility remains that variation in IGF1 could alter insulin secretion or action as well as childhood growth.
15 Finally, statins are also known to interfere with the synthesis of ubiquinone (CoQ10), which could independently alter insulin secretion.
Previous study in European samples showed the glucose-raising allele of MADD rs7944584 was associated with elevated fasting proinsulin without altering insulin secretion [21], suggesting this locus was associated with insulin processing defects.
E2 also acutely suppressed hyperglucagonaemia without altering insulin secretion, leading to normalisation of blood glucose.
In HOMA2, changes in β-cell function, altering insulin secretion, are entirely attributed to changes in Vmax and Km in the basal state (11).
In the β-cell, BAD alters glucose levels by altering insulin secretion by at least two different mechanisms depending on its phosphorylation status – an interaction with glucokinase and by its proapoptotic actions.
Applied together, targeted and nontargeted metabolomics followed by pathway analyses point to metabolic cascades associated with maternal glycemia and shed light on factors underlying maternal insulin resistance, altered insulin secretion, and resulting hyperglycemia.
