The phrase "alter host gene" is correct and usable in written English.
It can be used in scientific or biological contexts, particularly when discussing genetic modifications or changes to the genes of a host organism.
Example: "Researchers are exploring ways to alter host genes to improve resistance to certain diseases."
Alternatives: "modify host gene" or "change host gene".
An indirect promoter targeting mechanism could provide an opportunity for the virus (via AC2) to alter host gene expression.
In addition, viral proteins such as HBV X protein can influence intracellular signal transduction pathways and alter host gene expression, contributing to malignant transformation of chronically infected hepatocytes (Farazi and DePinho, 2006; Chemin and Zoulim, 2009).
An indirect promoter targeting strategy, via AC2-host dsDNA binding protein interactions, and inactivation of SnRK1.2-mediated defense responses could provide the opportunity for geminiviruses to alter host gene expression and in turn, reprogram the host to support virus infection.
An indirect promoter targeting strategy, via AC2-host dsDNA binding protein interactions, might provide the opportunity for geminiviruses to alter host gene expression and in turn, reprogram the host to support virus infection.
Given the frequency and scattered genomic distribution of ERVs and the influence of their LTRs on the expression of neighboring genes, ERVs have high potential to promote oncogene expression or to alter host gene expression networks to favor tumor development [ 25].
Drosha cleavage of intronic pri-miRNA precedes splicing, and the insertion of a miRNA hairpin into an intron does not alter host gene splicing or stability, indicating that a single transcript could give rise to both miRNA and mRNA [ 48, 51, 52].
This study demonstrates the value of a co-ordinated approach to assessing the roles of the EBV genes and proteins, showing extensive co-operation between the EBNA3s in altering host gene transcription, and identifying a number of genes consistently modulated by EBV, which may play key roles in effecting the biological imperatives of the virus.
In the present study we show that S. aureus infection altered host gene expression in a strain dependent as well as temporal manner.
Preliminary results indicated that PRRSV modulates the host immune responses and alters host gene expression.
It is a multifunctional oncoprotein that alters host gene expression by activating a plethora of cytoplasmic signal transduction pathways (e.g., NF-κB, Src, Ras, AP-1, AP-2, PI3K/Akt, Jak/STAT, Smad and Wnt) [ 1, 2].
Thus, the ability of effectors to suppress plant defenses, alter host cells or gene function, can directly impact pathogen fitness.
