The phrase "alter cell membrane" is correct and usable in written English.
It can be used in scientific or medical contexts when discussing changes or modifications to the structure or function of a cell's membrane.
Example: "Certain chemicals can alter cell membrane permeability, affecting the transport of substances in and out of the cell."
Alternatives: "modify cell membrane" or "change cell membrane".
FFAs are also believed to inhibit enzyme activity, impair nutrient uptake, and alter cell membrane permeability [ 8].
When occurring around cells, this process, referred to as non-thermal effects, is reported to alter cell membrane activity, vascular wall permeability, and facilitate soft tissue healing [ 12].
Consequently, their amino acid composition, amphipathicity, cationic charge, and molecular size allow cytotoxic peptides to adhere to and penetrate the phospholipid bilayer of the cell membrane, thus forming trans membrane pores, which will alter cell membrane permeability and cause that cell to undergo apoptosis [ 2, 3, 16].
In relation to this, hydrophobic compounds can alter cell membranes (Heipieper and Bont 1994; Whyte et al. 1999), including cell surface hydrophobicity, which enhances adhesion of cells to hydrocarbons in the water-hydrocarbon interface (Scott and Finnerty 1976; Rosenberg 1991; Baldi et al. 1999) and transportation through the cell membrane.
An altered cell membrane is suggested to be an underlying factor based on the type of neuromuscular findings.
At the cellular level, the effects of orexin accounted for evident argyrophilic reactions (neurodegeneration phenomena) including altered cell membrane and loss of cytoplasmic architecture in most amygdalar and hippocampal neuronal fields, while in the presence of flunitrazepam these reactions resulted to either be unappreciable or absent.
These results obtained in normal hearts will provide a base line for future studies of the metabolism of postischemic hearts which may have altered cell membrane permeability and/or enzymatic activity.
Further conclusive studies are needed to conclude the relation of antibacterial activity with ZnO-MFs as altered cell membrane permeability and intracellular metabolic system in bacterial cells caused by ZnO-MFs cannot be visualized by Bio-TEM images [30].
SigF mutants have reduced lethality in both mouse [28] and guinea pig models [30] of TB and also have altered cell membrane properties [31], [32] whereas sigJ is dispensable for in vivo growth but appears to be involved in protection against hydrogen peroxide [29].
The changes in peak Cho can reflect altered cell membrane synthesis and degradation and might correspond to glial cell proliferation (18, 19).
Overall, we observed age-dependent differences in PC, SM, AC, and AA levels that might be linked to altered cell membrane composition, mitochondrial metabolism, and counteracting oxidative stress.
