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Discover LudwigThe phrase "almost completely deficient" is correct and usable in written English.
It can be used to describe something that is lacking to a significant extent, but not entirely so.
Example: "The report was almost completely deficient in providing the necessary data to support the conclusions."
Alternatives: "nearly entirely lacking" or "practically void of".
Exact(2)
Third, some mutant channels (T187I and all truncated variants) were non-functional because of almost completely deficient maturation/trafficking (Figs. 1 and 6).
Here, we designed our thermotolerance assays to maximize differences in survival among strains varying in Hsp70 copy number and expression and have found the Hsp70- strain almost completely deficient in basal and inducible tolerance of severe heat shock.
Similar(58)
Although we did not observe a distinct defect in the degradation of damaged mitochondria in p62 KO MEFs, we noticed that the perinuclear clustering of depolarized mitochondria was almost completely inhibited in p62 deficient MEFs (Fig. 6A C).
Consistent with what we described above for the pandemic isolates, this toxicity towards the amoeba was almost completely abrogated in hemolysin-deficient mutant derivatives (Supplementary Fig. 5c).
In fact, the lung phenotypes are so pronounced that re-epithelialization and neutrophil recruitment into the alveolar space are almost completely abrogated in MMP7-deficient mice [3] [6].
In experimental animals, neointima formation was almost completely absent in midkine-deficient mice relative to control mice [ 2].
However, the C3 deposition was almost completely abrogated when the C2 deficient serum was applied (Figure 8B).
However, this endotoxin-induced systemic HMGB1 accumulation was almost completely abolished in IFN-γ-deficient mice (Fig. 2C), supporting an important role for IFN-γ in endotoxin-induced HMGB1 release.
Interestingly, G6PD reintroduction in some types of human cancer cells significantly rescues the growth of TAp73-deficient cells and almost completely restores the proliferation of human lung cancer H1299 cells expressing p73 siRNA (small interfering RNA) (Du et al., 2013; Jiang et al., 2013b).
LPCAT activity was almost completely absent in tissues of LPCAT3-deficient mice.
The mutations were almost completely abolished in the pol V-deficient strain, suggesting that this SOS polymerase is likely responsible for the error-prone translesion synthesis.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com