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Importantly, in addition to ChIP-on-chip approaches, two genome-wide location analyses utilized ChIP-PET and the sensitive ChIP-DSL approaches; all demonstrated no evidence for ERα recruitment to PCNA.
Densitometric analyses utilized TotalLab software (Amersham Biosciences, Freiburg, Germany).
Data analyses utilized GraphPad Prism 5.00 software (GraphPad, San Diego, CA, USA) and values at P<0.05 were deemed significant.
Statistical analyses utilized GraphPad Prism 4.00 software (GraphPad, San Diego, CA), with Student's paired t tests or ANOVA with the Bonferroni post hoc correction for multiple comparisons along with Spearman's correlation analyses.
Graphs and statistical analyses utilized Prism (GraphPad).
The multilevel analyses utilized 218,589 observations on 31,644 individuals for the cross-sectional analyses examining self-rated health and 181,830 observations on 25,505 participants for the longitudinal analyses.
The MS data were collected for the mass range of m/ z 50−1500, and MS/MS data analyses utilized Micromass software (MassLynx) version 3.5.
This hypothesis is derived from cladistic analyses utilizing 18S rDNA sequences [ 2- 4].
All analyses utilized a significance level of P < 0.05 and were performed using Prism (GraphPad).
All analyses utilized SAS v9.2 statistical software (SAS Institute Inc Cary, NC, USA).
All analyses utilized a two-sided P-value of 0.05 for significance.
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