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Measurements of zeta potential and algal cell surface architecture were performed to reveal the mechanisms of action of ferric chloride and chitosan as pre-flocculants.
For the second mechanism, the addition of GSH did not inhibit the attachment of Ag-ENs to the algal cell surface unlike what was observed by Li et al. [33], as part of the Ag-ENs already entered the cells.
They showed that algal cell surface glycan ligands, such as α-mannose/α-glucose and α-galactose play a role in recognition during initial contact at the onset of symbiosis [ 64].
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The adsorption of NP to algal cell surfaces (Fig. 2f) could accelerate their sedimentation (Fig. 2g), which forces pelagic consumers to invest more energy in collecting their food near the sediment [195].
Algal growth inhibition, cell surface hydrophobicity, oxidative stress, MWCNT-cell agglomeration, and cell morphology change and internalization of MWCNTs were assayed to investigate individual and combined toxicities of MWCNTs and DOMs, and mechanisms underlying different effects of DOMs on the toxicity of MWCNTs were specifically addressed.
The potential of the algal cell is increased, and the surface charge is neutralized (Henderson et al. 2008).
It is because the reducing agent, TCEP·HCl, released the PFDT layer through breaking the disulfide bond, and therefore detached the algal cells from the surface.
Bioaccumulation of arsenic, as well as, surface adsorption on algal cell was found considerably low.
These groups have tremendous capacity of metal ion binding on the surface of the algal cell wall.
Second, nanoparticles within the diffusion layer of algal cells or attached to the cell surface may dissolve and thereby provide additional metal ions directly to the algae.
Occasionally, cell surface material of algal cell was stained with ConA.
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