The phrase "alcohol aversion" is correct and usable in written English.
It can be used to describe a strong dislike or avoidance of alcohol, often in a medical or psychological context.
Example: "After his treatment, he developed a significant alcohol aversion that made it difficult for him to be around social gatherings where alcohol was present."
Alternatives: "alcohol intolerance" or "dislike of alcohol".
Mr. Edwards's first case as a plaintiffs' lawyer was for a man who had overdosed on a drug used in alcohol aversion therapy.
The researchers then used lab rats to understand which hormones were involved, showing that GLP-1 is implicated in inducing alcohol aversion while ghrelin can restore the rats' appetite for alcohol.
The plaintiff in the case, Howard E. G. Sawyer, was disabled as a result of what Mr. Edwards said was an overdose of a drug used in alcohol aversion therapy.
Activation of ALDH2 has been shown to benefit cardiovascular outcome following periods of ischemia and renewed interest in specific inhibition of ALDH2 has application for alcohol aversion therapy, and more recently, in cocaine addiction.
Aversion to alcohol was assessed using the alcohol aversion model of Garver et al. (2000).
N,N-Diethyldithiocarbamate besides being a main metabolite of disulfiram, the drug used over half century for alcohol aversion therapy, has diverse applications, also as pharmacological agent.
Our proposed strategy, based on the above principles, appears to satisfy the requirements for a potentially successful alcoholism therapy, one acting simultaneously on alcohol consumption through aversion, on anxiety and depression, and possibly also reward, through provision of serotonin and on hyperexcitability through decreased NMDA receptor function.
Importantly, LPS-triggered signaling through this complex has been recently reported to increase ethanol consumption and to alter certain aspects of alcohol reward and aversion in mice [ 46].
As mentioned earlier, LPS-triggered signaling through this complex has been recently reported to increase ethanol consumption and to alter certain aspects of alcohol reward and aversion in mice [ 46].
Carbidopa alone (Fig. 4b) caused aversion to alcohol, decreasing alcohol consumption by 30 33% consistently at 2 4 h (P = 0.0162 0.0017), in comparison with saline-treated controls.
CONCLUSIONS: The principal difference between the Sprague-Dawley and P rats was that the P rats did not show the normal aversion to alcohol after forced exposure to alcohol that the Sprague-Dawley rats showed.
