The phrase "ais while the" is not correct in written English.
It appears to be a typographical error or a fragment that lacks clarity and coherence.
Example: "The project was delayed, ais while the team was waiting for approval."
Alternatives: "as the" or "while the".
The fast one was an electrotonic projection of the spike generated in the AIS while the slow one corresponded to the antidromic spike invasion to the axon hillock and soma [42], [43].
Interestingly, we found that α MNs (identified as Err3-/NeuN+; data not shown; [ 35]) are divided into two subpopulations: 15% of them had a dendrite-derived AIS, while the remaining 85% had a soma-derived AIS (Table 1).
A total of 512 mRNAs were upregulated in AIS, while 34 mRNAs were downregulated.
This is probably because AAH sometimes involves thickened alveolar septa similar to those of AIS, while AIS is sometimes associated with less thick alveolar septa similar to those of AAH.
We found that a mean CT value less than −520 HU indicates AAH or AIS, while a mean CT value greater than or equal to −520 HU indicates MIA.
Approximately half the children presenting with AIS have a predisposing cause 14 (symptomatic AIS) (fig 3), while the remainder were previously well (cryptogenic AIS); around 80% of both groups have abnormal arterial imaging.
We have clustered the injuries per body region as described in the abbreviated injury scale (AIS) [ 45], while the injury of the face was added to the head/neck region.
While AnkG also clusters KCNQ2/3 at the AIS [ 17], the mechanisms controlling expression of Kv1 channels at the AIS remain unresolved.
Among the MNs expressing Kv1.1 at P5, 16.8% expressed Kv1.1 at a low level in both AIS and JXP-AIS, but did not yet express Caspr in their presumptive para-AIS; while 50.5% of MNs expressed Kv1.1 at a low to moderate level in both AIS and JXP-AIS and also expressed Caspr at the para-AIS.
At P3, 83.3% of MNs (n = 38) were still not expressing Kv1.1 in their AIS or JXP-AIS, while 16.7% showed a weak expression of Kv1.1 in their AnkG+ AIS, but did not yet express Caspr (as for the following stages/populations, a representative example is shown, in Figure 7E-H).
Despite many controversies and unanswered questions about AIS, the most difficult one is the aetiopathogenesis of AIS.
