Sentence examples for aip 1 from inspiring English sources

Patients with type II AIP (1 patient histologically confirmed) were excluded from the analysis.

The hyperphosphorylation of CaMKII targets could be prevented by Ran (10 µmol/L), TTX (2 µmol/L), and AIP (1 µmol/L), but not by H89 (5 µmol/L; Figure  6 A and B, P < 0.05 each).

Isolated CMs were stained and measured as annotated above with the following differences: Fluo 3 AM loading buffer either contained Ranolazine (10 μmol/L), AIP (1 μmol/L), H89 (5 μmol/L), KBR (0.1 μmol/L), or no active agent.

Of note, an inhibition of PKA (H89 5 μmol/L) as well as an inhibition of both kinases (H89 5 μmol/L + AIP 1 μmol/L) also attenuated the ATX-II induced increase of CaSpF and significantly reduced the calculated SR-Ca2+-leak compared with sole ATX-II treatment.

Interestingly, simultaneous CaMKII inhibition (AIP, 1 µmol/L, Figure  2 A– D) as well as an inhibition of NCX (KBR, Figure  3 A D) also prevented the ATX-II-induced increase of CaSpF (P < 0.001 each) and SR-Ca2+-leak (P < 0.05 and <0.01, respectively), suggesting that CaMKII is activated via NCX-dependent Ca2+ overload.

Colonic manifestation has seldom been reported as a complication in cases of type1 AIP [ 1, 2].

The CR in the scenario of aggregation by geometric mean was markedly lower for AIJ than for AIP (CR AIJ: 0.0045; CR AIP: 0.0490), although only participants with a CR ≤ 0.1 were included for the AIP.

The organ that is most commonly involved in IgG4-related sclerosing disease is the pancreas; such involvement is termed autoimmune pancreatitis (AIP) [1].

In addition, delayed enhancement of a peripancreatic capsule is highly suggestive of AIP [21].

Renal involvement is frequently seen with AIP, and renal parenchymal involvement is noted around 30% of patients with AIP [43].

By using the median (CR AIJ: 0.0683; CR AIP: 0.0674) or mean scenario (CR AIJ: 0.0745; CR AIP: 0.0587), the CRs were similar, but still much higher than the CR from AIJ by geometric mean, as expected.