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The observed changes of the lamellar structure during aging were found by TEM to be related to the following defects present in the lamellar structure prior to aging: low angle grain boundaries, misoriented lamellae, lamellar interfaces and lamellar colony boundaries.
In that system, several hundred promoters, including those involved in aging, were found to display differential cycling of O-GlcNAc [ 52].
At the genome-wide level, however, CR and aging were found to have weak association, which was stronger in heart relative to liver, muscle and central nervous system.
Different patterns of aging were found among regions: Aragon, Catalonia and the Basque Country showed the greatest ageing, with more than 34% of their populations being over 50 years of age.
Despite the shortening of processes, no significant differences with aging were found in the complexity of the branching pattern of the microglia processes (number of branch points per process) and/or number of processes per cell (Fig. 1F,G).
No markers of accelerated aging were found in the other two models, leading to the speculation that, rather than increased p53 activity per se, altered functionality of the p44 p24 /p53 complex might explain those premature aging phenotypes (Mendrysa et al., 2006).
Similar(54)
Aging was found to significantly increase the spatial variations of the sample properties.
Electroless aging was found to be inevitable for all the carbon felts used.
The effect of aging was found to be significantly different in the two alloys.
No effect of layer thickness on the bitumen aging was found.
Overall an effect of aging was found that interacted significantly with stimulation (F=6.2, P=0.018).
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