Sentence examples for aging in vitro from inspiring English sources

The data also provide biological insights into which cell characteristics (proliferation and adhesion, for example) are functionally clustered and either change or remain constant with aging in vitro.

Though lack of a clear correlation between organismal aging with cellular growth viability, the study of mammalian cell aging in vitro has enormous potential for telling us how human aging works [1].

The authors of the paper argue that neither replicative aging in vitro nor cell donor's advanced age prevent cells from dedifferentiation into iPSCs.

Several factors may contribute to these divergent results, including developmental stage (embryonic vs. adult), tissue of origin (lung vs. skin), aging in vitro, transient overexpression of the protein, and/or interactions with the genetic background of distinct donors.

However, when cultured for over 2.5 months (a condition mimicking aging in vitro), DAn differentiated from patient-specific iPSC developed evident signs of neurodegeneration, including fewer and shorter neurites and a significant increase in apoptotic cells.

Several model systems for cellular aging have been developed, and a representative selection of these models was applied for the present study: replicative senescence, a process of cellular aging in vitro that is primarily driven by telomere erosion, is a classic experimental system considered relevant for the aging of tissues with high regenerative potential (von Figura et al., 2009).

Samples were subsequently aged in vitro at a constant temperature in artificial saliva supplemented with food colouring for two cycles of 14 days each.

We observed that immediately fertilized eggs from the second spawn showed also a high loss in development potential like eggs from the first spawn aged in vitro for 3 hours (Fig. 4A B).

Neutrophils were isolated on a MACS MS separation column and aged in vitro as described above.

when they are aged in vitro (Sanchez-Danes et al., 2012; Isobe et al., 2014).

To gain further insight into the role of the miR-17 92 miR-17 92n aging of clusteryocytes, neonatal rat cardinmyocytes (NRCMs) were agingin vitrof and miRNA levels were determined.