The phrase "affecting the excitability" is correct and usable in written English.
It can be used in contexts related to biology, neuroscience, or psychology when discussing how certain factors influence the responsiveness or reactivity of cells or organisms.
Example: "The study focused on how various neurotransmitters are affecting the excitability of neurons in the brain."
Alternatives: "influencing the reactivity" or "modulating the responsiveness".
Baclofen did not affect the excitability of peripheral human axons which is consistent with reports from isolated human DRG neurones [4].
Indeed, high cortical activity levels could cause slow depolarizations and affect the excitability properties of the neuron, and therefore its information transmission properties, via adaptive threshold dynamics.
However, future studies may use the presented in this study protocols to answer an important question as to how a concurrent activation of CA1 pyramidal neurons and CA1 interneurons by physiological concentrations of choline and 1 5 µM PNU-120596 affects the excitability of pyramidal neurons and thus, the hippocampal output.
While both LICI and SICI directly affect the excitability of corticospinal neurons, there are also interactions between them, as LICI appears to reduce SICI, likely via GABA-mediated inhibition of GABA-release [ 39, 43].
We found that vestibular activation affected the excitability of both EVC and V5/MT, but this does not necessarily prove that vestibular signals have a specific functional interaction with visual cortex.
They generally interact with metabotropic or G-protein-coupled receptors, whereas neurotransmitters are chemical substances that affect the excitability of neurons acting as mediators of the neurotransmission through synapses.
Therefore considering recent modelling data suggesting that the propagation of the action potential within SN neurons is highly energy dependent based on the complexity of the axonal arbour of these neurons, a decrease in the production of ATP will affect the excitability of these neurons which would further increase their vulnerability (Pissadaki and Bolam, 2013).
Because changes in motoneuronal excitation following M1 excitation can last as long as 80 to 100 ms, it is apparent that at the C-T intervals used in this study, there was ample time for TMS to affect the excitability state of FRA interneurons that produce polysynaptic reflex actions on α-motoneurons.
As a result, lamotriqine that increased Ih current preferentially reduced dendritic excitability, while minimally affecting the somatic excitability of CA1 pyramidal neuron [25].
