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Although this paper focused on differential negative effects of family adversities (i.e., dual risk), the described mechanisms might work both ways, in such that the same mechanisms might lead to differential pro-social behavioral outcomes under environmental enrichment (i.e., differential susceptibility, Belsky 2005).
They helped me realize that the adversities I had overcome both as a child and an adult, as well as my "unconventional methods" of achieving success in my life, were tools that I could use to help kids who suffer great adversity, or lack opportunity.
For all those fighting the good fight let me share my story of overcoming one of the greatest adversities I've ever endured: coming back from a spinal injury that very nearly ended my skiing career.
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Currently, information concerning omic × environment interactions might be discussed qualitatively in assessments as supporting information, but, to date, such data have not been widely defined in regard to adversity (i.e., adverse or not adverse).
In this study we investigate the impact of very early life adversity (i.e. exposure to depression in utero) on adulthood risk factors for age-related disease.
However, there is limited research into the effects of very early life adversity (i.e. exposure to depression in utero) on inflammation.
This study examined the differential impact of early life adversity (i.e., experienced prior to age 18) and adversity experienced during adulthood on syndemic production among MSM.
However, because most studies test the cumulative aversive effects of genotype and environment (i.e., dual risk), rather than differential susceptibility or vantage sensitivity, we solely reviewed findings involving environmental adversity (i.e., the "for worse" part).
For example, Dmitrieva et al. (2011) found that the gender-specific direct effects of the DRD4 on externalizing behavior were explained by differences in exposure to family adversity (i.e., poor parental monitoring and exposure to violence).
Most notably, our findings provide initial evidence that childhood adversity (i.e. abuse) and both life events and recent cannabis use may combine synergistically to increase the likelihood of psychotic experiences.
All children had experienced at least some degree of adversity (i.e., no child obtained the lowest possible score on all 11 adversity variables), which is important given that by definition, the presence of adversity is required to be able to exhibit resilience (e.g., [ 16]).
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CEO of Professional Science Editing for Scientists @ prosciediting.com