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Obesity greatly increases a child's risk in adulthood of developing and dying from serious chronic conditions such as cardiovascular disease, type 2 diabetes mellitus, and certain cancers.
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Childhood overweight and obesity tracks into adulthood, increasing the risk of developing future chronic disease.
If this view is correct, then AD is a disease most people are in the process of developing throughout adulthood.
Obesity frequently tracks from childhood into adulthood and increases the risk of developing chronic diseases, including type 2 diabetes mellitus, cardiovascular disease and some types of cancer [ 4].
Babies affected by SGA on reaching adulthood are at greater risk of developing cardiovascular disease, hypertension, and non-insulin dependent diabetes [ 4, 5].
Preventing or at least delaying the onset of abdominal obesity in young adulthood may lower the risk of developing diabetes through middle age.
These babies on reaching adulthood are at greater risk of developing cardiovascular disease, hypertension, and non-insulin dependant diabetes [ 4, 5].
While patients who received chest radiation for Hodgkin's lymphoma during adulthood have established increased risks of developing future breast cancers, hence requiring closer surveillance, the risks of radiation-associated second cancer risks in other sites are likely much lower.
Globally the prevalence of child overweight and obesity is increasing, with 9% of children aged 0 5 years expected to be overweight or obese by the year 2020. 1 Overweight and obesity in childhood tracks into adulthood, 2 increasing the risk of developing a number of chronic health conditions.
The probability of developing obesity in adulthood was slightly more pronounced with similar increases in the likelihood being found across the 3 studies using this outcome (OR 1.4-2.27) [20 22].
Prospective epidemiological studies indicate that dyslipidaemia early in life [ 22, 23] or during adulthood [ 24] raises the risk of developing type 2 diabetes later in life, but such associations may be driven by obesity [ 22] rather than a lipid-specific genetic defect.
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