The phrase "administration exposure" is not commonly used in written English and may require additional context to be clear.
It could be used in discussions related to the visibility or awareness of administrative actions or policies, particularly in a governmental or organizational context.
Example: "The report highlighted the need for greater administration exposure to ensure transparency in decision-making processes."
Alternatives: "administrative visibility" or "administrative awareness".
However, the risks of contrast administration, exposure to ionizing radiation, and cost are all limiting factors [14 18].
Population or other pharmacometric models are a useful means to describe, succinctly, the relationships between drug administration, exposure (concentration), and downstream changes in pharmacodynamic (PD) biomarkers and clinical endpoints, including the mixed effects of patient factors and random interpatient variation (fixed and random effects).
The brain can sustain physical injury and also, from the administration or exposure to chemicals.
Blood clearance of DEI was as much as 10-fold lower than that of ISO in animals and after oral administration, metabolite exposure exceeded that of parent by as much as 14-fold.
The post-hoc questionnaires used in this study have disadvantages due to their administration after exposure; these disadvantages include recency effects, anchoring effects and inaccurate recall [ 55].
Specifically, the time for the PD MPS and mixing chamber to become well-mixed following drug administration, drug exposure, and the elimination of total drug in the platform.
Intriguingly, G-CSF administration after exposure to 4 Gy TBI led to a much higher level of ROS in HSCs than that in the 4 Gy TBI group and G-CSF group.
After prazosin administration, CAPs exposure was not associated with statistically significant changes in any hemodynamic parameter (Table 3), but we had limited statistical power to detect effects of this magnitude in this sample.
In contrast to the enhanced inflammatory response seen during steatohepatitis following chronic ethanol administration, acute exposure to ethanol in vivo suppresses various inflammatory responses (e.g., leukocyte recruitment and endothelial cell activation) (Saeed et al. 2004).
It is hypothesized that an altered metabolite profile of circulating insulin prevails after subcutaneous administration due to exposure of insulin to epidermal proteases.
Increased awareness towards nanotoxicity, studies have been reported on the in vivo toxicity of CuNPs and ZnONPs for intranasal instillation [22, 23], intratracheal instillation [24, 25] and oral administration [26 28], dermal exposure [29, 30].
