Sentence examples for administered methyl from inspiring English sources

For this purpose, we administered methyl levodopa repeatedly to a rat model of Parkinson's disease with unilateral 6-hydroxydopamine 6-OHDA -inducedd lesion of the nigrostriatal dopamine pathway.

This is supported by the report that microsomes from the livers of rats administered methyl eugenol at doses of 30 300 mg mg/kg autoinduced 1′-hydroxylation of methyl eugenol, whereas those of rats administered 10 mg/kg did not (Gardner et al. 1997).

Finally, another important consideration is that the rodent bioassay was conducted using oral gavage as a means of administering methyl eugenol.

Evaluating the impact of route by administering methyl eugenol in the diet of rats and mice would add important information to our understanding of chronic dietary exposure to methyl eugenol.

Light emission was readily apparent in mice administered with iLH2  6 methyl ester.

We also used an alternative method, by administering 3-methyl adenine (3-MA), an autophagy inhibitor, via the tail vein in mice, and our data showed that alveolar macrophage autophagy was decreased, as evidenced by the quantitation of punctuate dots using confocal microscopy and by LC3 immunoblotting.

We repeated these experiments (Supporting Information Figure S3) and confirmed that methyl-β-cyclodextrin treatment also increases the PS-induced activity of mouse TRPM3 channels, while additional cholesterol (administered as a methyl-β-cyclodextrin/cholesterol complex) reduces mouse TRPM3 channel activity, as demonstrated previously for human TRPM3 channels (Naylor et al., 2010).

Diabetic animals were treated with single dose of 5 mg/kg AZL suspended in 1% carboxy methyl cellulose, administered orally by gavage, starting the 4th day of STZ treatment (n = 12) daily for a period of 12 weeks.

Silverstein et al. (abstract 130) administered the noncompetitive N-methyl d-asparate receptor antagonist used for treatment of Alzheimer's disease immediately after subjecting rats to glucose 10 15 mg/dl for 90 min; compared with untreated rats, hypoglycemia-induced cortical neuronal damage decreased 35%.

After determination of control responses to adenosine a 10-mg/kg dose of the nitric oxide synthase inhibitor N ω-nitro-L -arginine methyl ester was administered into the femoral vein; the dose-response curves to adenosine were then determined again.

One hour after, 3 ml of a semi solid test meal (based on methyl cellulose) was administered to the mice.