The phrase "adjustment to exercise" is correct and usable in written English.
It can be used when discussing changes or adaptations made in response to a new exercise routine or physical activity.
Example: "After a few weeks of training, I noticed a significant adjustment to exercise was necessary to prevent injury."
Alternatives: "adaptation to exercise" or "modification for exercise".
Further studies are warranted to better clarify the role of mechanoreflex in the cardiovascular adjustment to exercise pressor reflex activation.
The "resetting" of the arterial baroreflex is essential to evoke and maintain an effective autonomic nervous system modulation and an adequate cardiovascular adjustment to exercise.
Adjustment to exercise is a complex phenomenon that could be briefly described as an increase in oxygen and energetic metabolites supply to working muscle.
An authority in the area of metabolic adjustment to exercise, Brooks developed the lactate shuttle theory, a fundamental biological mechanism that allows muscles to take up lactate via transporter proteins that "shuttle" lactate within and between cells.
Decades of research in the area of metabolic adjustments to exercise led to articulation of two working hypotheses: the "Lactate Shuttle," and the "Crossover Concept". Research on the Lactate Shuttle was to elucidate the pathways and controls of lactate formation and removal before, during and after exercise.
These experiments demonstrated that metabolic reflex originating in skeletal muscle can mediate cardiovascular adjustments to exercise, the so called "metaboreflex".
However, HR and blood pressure responses were attenuated as compared to a normal exercise, thereby suggesting that afferent feedback from the muscle is essential in normal cardiovascular adjustments to exercise [ 9, 124, 125].
In detail, cerebellum, insula, anterior cingulate cortex, medial prefrontal cortex, hippocampus, thalamus, and possibly others have all been demonstrated to be potentially involved in this mechanism and all may take part in the circulatory adjustments to exercise [ 11, 58– 63].
Therefore, early research suggested that the arterial baroreflex was "switched off" as it was unnecessary for the cardiovascular adjustments to exercise or alternatively that the sensitivity of the reflex was significantly decreased during exercise to increase both HR and arterial blood pressure [ 19, 70, 72].
