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DP reduced gene expression of VCAM-1 (Fig. 5Ci), a vascular adhesion molecule promoting integrin-dependent adhesive interactions of T cells with venules, by 60%.
Selectins are vascular cell adhesion molecules (VCAMs) involved in adhesive interactions of leukocytes, platelets, and endothelial cells that mediate leukocyte trafficking and hemostasis.
Disruption of F-actin increases cell adhesion, whereas disruption of tubulin reduces adhesive interactions in vivo [34].
The laboratory line A4u binds moderately to ICAM-1 [36] with little stationary adhesion under flow whereas the main adhesive interactions were of the rolling type.
These multistep events require complex adhesive interactions between ligands on cancer cells and cellular adhesion molecules and other substrates present in the tumor microenvironment [21], [34].
While aglycemia challenged gliovasculature did not affect lymphocyte adhesive interactions, both hypoxia and OGDR significantly increased lymphocyte adhesion in vitro compared to control (normal).
By contrast, for NL3A2 presence of the AS6+ insertion in NRX1alpha resulted in slightly increased adhesion and isoforms containing AS6 but lacking AS3 showed the strongest adhesive interactions.
Characterization of adhesive interactions between human endothelial cells and megakaryocytes.
Figure 3: Adhesive interactions between the DCL and EVL during tissue spreading.
Kulkarni, S. et al. Techniques to examine platelet adhesive interactions under flow.
Evidence that Myc activation depletes the epidermal stem cell compartment by modulating adhesive interactions with the local microenvironment.
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