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The limited adherence shown to clinical guidelines has been termed clinical inertia.
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According to the World Health Organization's multidimensional adherence model (MAM), medication adherence is shown to be multifactorial [ 1].
Results for self-reported and electronically monitored adherence are provided in Table 2. Overall, average electronic adherence was shown to be 0.76± 0.23 and self-reported adherence was 0.78± 0.78±
Hence, we could not analyze adherence in this setting as in comparable African settings [ 14, 24, 25, 46], where good adherence was shown to be associated with improved outcome under cART.
However, two dscam genes (dscam2 and dscam5), known to be involved in cell adherence, were shown to be downregulated in this study [87], [88].
Medication adherence was shown to improve in the other study on electronic monitoring-based intervention.
No one theoretical model underpinning exercise adherence was shown to be superior and the interventions that incorporated motivational strategies showed promise.
Medication Adherence Report Scale (MARS) Measuring self-reported non-adherence as opposed to self-reported adherence has been shown to correlate more closely to objective adherence measures such as pill counts [ 21 ].
Poor adherence has been shown to be linked to patients' beliefs about medicines with a potential impact on adherence.
Low levels of adherence have been shown to increase risk of disease progression burden on services and treatment cost [ 4– 8].
Both QOL and adherence have been shown to be associated with HIV RNA levels, HIV disease stage, and symptoms.
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