Sentence examples for adherence of pathogens from inspiring English sources

Probiotic bacteria benefit the host by adhering to the gut epithelium, stimulating host immune response, inhibiting epithelial and mucosal adherence of pathogens and producing antimicrobial substances [7].

Adherence of pathogens to host cells is pre-requisite for cell invasion [30], [31].

Several in vitro studies suggested that probiotic adhesion may interfere with the adherence of pathogens, exerting a barrier against pathogen colonization through competitive exclusion mechanisms [ 39, 40].

The strain produces lactic acid hydrogen peroxide, and bacteriocin and adheres well to epithelial cells, and thus significantly inhibits the growth and adherence of vaginal pathogens [ 16].

Immunofluorescence labeling further revealed greater adherence of these pathogens to the colonic crypts of infected Il22ra1−/− mice.

It may be noted that since the adherence of the pathogen to the gut epithelium is the foremost stage of the disease process, inhibition of adherence could be a very important aspect in the antidiarrhoeal activity of the plant.

It is not within the scope of our study to give a comprehensive explanation for this finding, but it can be speculated whether this reflects a pro-inflammatory situation or a concomitant infectious disease that impairs the protective mucosal barrier and could thus promote a more effective adherence of the pathogen.

We examined FlpA, a C. jejuni Fn-binding protein composed of three FNIII-like repeats D1, D2 and D3, to identify the interactions required for cellular adherence on pathogen-induced host cell signaling.

The presence of oral microorganisms in a biofilm within endotracheal tubes may have significance beyond facilitating the adherence of potential respiratory pathogens.

Binding specificity of lectin to N-acetylgalactosamine, mannose and sialic acid might have played the key role in inhibition of Shigella dysenteriae to HT29 cells, as N-acetylgalactosamine, sialic acid (pathogen factor) and mannose (host factor) are involved in the initial adherence of Shigella dysenteriae during Host-pathogen interaction.

Thus, the evidence supports that c0139r mediates cell aggregation as a autotransporter encoding gene, and moreover, enhances the diversity of adherence methods of this pathogen and widens its target cell spectrum.