The phrase "address the expression levels" is correct and usable in written English.
It can be used in contexts related to scientific research, particularly in fields like biology or genetics, where one discusses the measurement or analysis of gene or protein expression levels.
Example: "In our study, we aim to address the expression levels of various genes involved in the disease."
Alternatives: "tackle the expression levels" or "evaluate the expression levels".
However, there have been no studies addressing the expression levels of TET2 in CLL.
Here, we investigated whether H2S played a regulatory role within the periodontal remodelling process by addressing the expression level of OPG/RANKL in hPDLCs.
To address this question, we compared the expression levels of cyclin D1 and the capacity to transit from proliferative growth to quiescence in KRIT1−/− and Lv-KRIT1 MEFs grown to post-confluence either in the absence or presence of the ROS scavenging agent N-acetylcysteine (NAC).
In order to address this question, we studied the expression levels of the 11 genes in kidney biopsy samples from subjects with three other common forms of primary GN: focal segmental glomerulosclerosis (FSGS), membranous nephropathy (MGN), and minimal change disease (MCD).
To address this question, we examined the expression levels of Bim, Bid and PUMA in HTLV-1-infected T-cell lines.
In order to address this, we looked at the expression levels of a number of TLR signalling-associated genes before and after LPS stimulation using a QRT-PCR-based array system (see Table 1).
To address these issues, we modulated the expression levels of TDP-43 in the Drosophila glia and found that the protein was required to regulate the subcellular wrapping of motoneuron axons, promote synaptic growth and the formation of glutamate receptor clusters at the neuromuscular junctions.
When these transgenic lines are available, we are going to addresses several issues: (i) how the expression levels of Mecr are regulated in vivo; (ii) the molecular and physiological mechanisms of the cardiac phenotype of the Mecr transgenic mice; (iii) whether the poor exercise performance of the Mecr transgenic mice is solely because of the overexpression of Mecr in cardiac myocytes.
To address the above issue, we measured the expression levels of several members of the TGFβ superfamily.
To address this question, we first compared the expression levels of HIF-1 proteins in HTLV-1-infected and non-infected T-cell lines.
