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Additionally, we discover and independently verify thirty-one previously unknown KIR alleles using methods we developed to accurately map and call the highly polymorphic HLA and KIR loci from exome capture data.
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Additionally, we discovered long-range contacts between gene bodies of exon-rich, active genes in all cell types.
Additionally, we discovered that XANM-ADSCs in immunodeficient mice had apoptosis rates similar to XANM-ADSCs-TGFβ1 over a short time course (7 days).
Additionally, we discovered that patients with a lower frequency of attacks (ie, HFEM; 8-14 headayse days per month) appeared to achieve a better response than those with a higher frequency of attacks (CM; ≥15 headache days per month).
Additionally, we discovered natural auto-control mechanisms, such as the sex-linked lethals causing distorted sexual ratios.
Additionally, we discovered probes targeting polymorphic CpGs that overlapped SNPs.
Additionally, we discovered private alleles in both populations (Fig. 2).
Additionally, we discovered 51 differentially expressed miRNAs in epithelial and stromal cells.
Additionally, we discovered that susceptibility to acetaminophen-induced liver damage was altered after knockdown of three gene candidates.
Additionally, we discovered other variables (i.e., admission status, age, emergency classification, procedure, and diagnosis) that independently affect data sufficiency.
Additionally, we discovered that the operation procedure has a significant impact on the refractory period in adult males.
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