Exact(7)
Additionally, defects in the ROS mediated killing of P. aeruginosa by murine CFTR-deficient alveolar macrophages have been recently reported by Zhang et al [14].
Additionally, defects in core fucosylation have been associated with abnormal lung development and an emphysema-like phenotype in Fut8 deficient mice [32], [33].
Additionally, defects in phagocytic processes related to corpse removal result of autoimmune diseases and impaired neuronal function [31], [38], [39] and several players involved in this process have been implicated in pathogenesis of infectious agents [40] [42].
Additionally, defects in CD154 (CD40 ligand) and CD40 may present in infancy with P. jiroveci pneumonia.
Additionally, defects in catecholaminergic neurotransmission due to a mutation in the vesicular monoamine transporter, cat-1, resulted in abnormal avoidance responses only at ΔT = 0.4°C.
Additionally, defects in anterior embryonic patterning have been associated with disruption of prechordal plate markers (Ang and Rossant, 1994; Weinstein et al., 1994; Filosa et al., 1997; Bachiller et al., 2000; Petryk et al., 2004; Nishioka et al., 2005; Warr et al., 2008).
Similar(53)
Additionally, defect densities such as vacancies or dislocations also remain constant.
Additionally osteochondral defects of 1.5 mm (Dahlin et al. 2014) and 2 mm (Chung et al. 2014) diameter on the femoral condyle have been used for the assessment of biomaterial strategies (Singh et al. 2013).
Additionally, laterality defects were observed in Bcor morpholino injected frogs, similar to those present in a subset of OFCD patients [21].
Additionally, these defects were also observed in U2OS cells (Supplementary Fig. S4A and B).
Additionally, multiple defects of the internal organs were found, i.e. renal dysplasia and intestinal atresia.
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