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It has additionally been postulated that EPA blocks the terminal stages of aracidonic acid synthesis from its precursors in vivo [ 31].
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The neutrophil has been postulated that is the responsible cell in the pathogenesis of TRALI.
Additionally, it has been postulated that elevated levels of TNF-α may increase soluble VCAM-1 in SLE [ 46].
Additionally, it has been postulated that therapeutic interventions and cardiovascular risk modification can influence myocardial blood flow or flow reserve of the heart [ 20– 20].
Additionally, it has been postulated that mammalian cells are able to selectively remove moderately aldehyde-modified proteins from their intracellular protein pools and that the proteasomal system is responsible for this activity [ 42].
Additionally, it has been postulated that during epithelial mesenchymal transition (EMT) a so‐called "cadherin switch" comes into effect, in which E‐Cadherin, typically expressed in epithelia, is replaced by N‐Cadherin, found mostly in mesenchymal tissue 58.
It has been postulated that 2-hydroxylated estradiol is anticarcinogenic.
It has been postulated that RLS may be caused by dysfunction in central dopaminergic pathways.
It has been postulated that iPS cells are generated exclusively from Muse cells [25].
It has been postulated that increasing the femoral head diameter may reduce the risk of dislocation.
Additionally, it was postulated that jasmonate itself may be transported over long distances to induce wound response [ 8].
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