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Interestingly, the more recently published reports on CDKN1A, PDK4, and IGFBP3 in addition could demonstrate cycling of the synthesis of mature mRNA.
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In addition, we could demonstrate that the combination of a proteasomal inhibitor and DMF superinduced the p21 expression.
In addition, we could demonstrate that both tinnitus and electromagnetic hypersensitivity are independent predictors for reduced discrimination ability.
In addition, we could demonstrate that the inhibition of the Ca response (but not the ERK one), was fully reversed when PD-1+ PBT were treated with SSG (Fig. 6 C and D).
In addition, we could demonstrate that α7-nAChRs are involved in regulation of bone remodelling.
In addition, we could demonstrate that tin2 was induced during biotrophic growth.
In addition, we could demonstrate the inhibitory effect of Sorafenib on the PI3K/Akt pathway.
In addition, we could demonstrate that both cardiac biomarkers hsTnT and NTproBNP were independently associated with CI-DCI.
In addition, we could demonstrate that patients with a high SOCS2 expression are more likely to experience biochemical tumor relapse.
In addition, we could demonstrate that IL-17, in synergy with IFN-γ, can induce the expression of iNOS, COX-2, and PD-L1 in MSCs.
In addition, we could demonstrate that hAPLN was associated with more severe histopathological renal findings as well as with development of ESRD in long-term follow-up.
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