The phrase "adapting gene expression" is correct and usable in written English.
It can be used in contexts related to biology, genetics, or molecular biology, particularly when discussing how organisms modify their gene activity in response to various factors.
Example: "The study focused on how environmental changes are influencing the process of adapting gene expression in plants."
Alternatives: "modifying gene expression" or "altering gene expression".
Overall, these data show that the pathogenic Bacillus strains have evolved ways to adapt gene expression in a more profound and extensive way in different atmospheric conditions.
mRNA decay is, however, an essential process that allows cells to continuously adapt gene expression to internal and environmental changes.
mRNA decay is an essential and active process that allows cells to continuously adapt gene expression to internal and environmental cues.
Sirtuins are highly conserved NAD+-dependent protein deacetylases and/or ADP ribosyltransferases that target histones, transcription factors, and co-regulators to adapt gene expression in response to the cellular energy state [ 5].
Among these, peroxisome proliferator activated receptors (PPARs) play a prominent role by acting as lipid sensors that cooperate in different organs to adapt gene expression to a given metabolic status (Desvergne et al. 2004; Evans et al. 2004; Feige et al. 2006).
Based on the two different regulation types: transcriptional and post-translational regulation, AADC is regulated by a quick acting, short- term mechanism, via regulation of the protein activity and in a slower longer lasting regulation, by adapting the gene expression [ 115, 116].
The dynamic nature of epigenetic modifications, as described by Adrian Bird, 'to register, signal or perpetuate altered activity states' through 'the structural adaptations of chromosomal regions' [ 5] allows RASF to adapt their gene expression to the highly reactive microenvironment of the rheumatoid synovium.
In the present study, SBE was adapted for gene expression profiling.
These findings were fine-tuned and adapted to gene expression time course data triggered by TGFβ1 and TNFα in SFB from RA and OA patients.
