The phrase "adaptation the selection" is not correct in written English.
It appears to be a fragment and lacks clarity, making it unusable without additional context or correction.
Example: "The adaptation the selection process underwent was crucial for survival."
Alternatives: "adaptation of the selection" or "selection for adaptation".
Sometimes, on the other hand, the term "adaptation" is reserved for traits that are "good for" their owners, that is, those that provide a "better fit" with the environment, and that intuitively satisfy some notion of "good engineering".[7] These two meanings of adaptation, the selection-product and engineering definitions respectively, are distinct, and in some cases, incompatible.
One of the important issues in developing biomarkers for clinical adaptation is the selection of appropriate cells or tissues from which to identify candidates and then develop biomarkers.
In order to increase the robustness of the search process and to facilitate its use by planners on different networks and operation conditions, the proposed method uses multi-operators and a mechanism for dynamic adaptation of the selection probabilities of these operators.
In adaptation to the selection pressure exerted by herbivores, plants evolved constitutive and inducible defense systems that appear to be tailored specifically to different aggressors [ 5, 6].
The selection pressure applied to the worm population was a key factor for worm adaptation, with the selection differential between worm genotypes and the proportion of the worm population exposed to the selection pressure (i.e. the frequency of the low WEC allele in sheep) being key parameters.
As in any perturbed biological environment, herbicide use has resulted in plant evolution and adaptation by the selection of genetic traits conferring phenotypic resistance (i.e., mechanisms protecting plants by a reduction in the herbicide damage) and allowing weedy plants to survive and reproduce in the presence of herbicides.
In this paper we do not present a language to concretely define the triggering of adaptations and the selection of the most appropriate reconfiguration.
In conclusion, repeated exposure of bacteria to certain microbicides in vitro can result in decreases in antimicrobial susceptibility that may be transient or sustained, probably resulting from temporary phenotypic adaptations or the selection of stable genetic mutations, respectively.
Of note, all clonally expanded cells recapitulated characteristics of the CMS-induced cells consistent with stochastic adaptation rather than the selection of a pre-existing subpopulation of MDA-MB-231 cells.
