The phrase "adapt the brain" is correct and usable in written English.
It can be used in contexts discussing cognitive flexibility, learning, or changes in mental processes.
Example: "To improve problem-solving skills, one must learn to adapt the brain to new challenges and perspectives."
Alternatives: "retrain the mind" or "modify cognitive processes."
Interactions with the external environment update and adapt the brain's functional architecture, particularly during critical periods of development, as exemplified by barrel formation, as well as on a continuous basis throughout life.
The hippocampus draws much interest as a model system because adult-born neurons adapt the brain to temporal events in external space including spatial learning and retention, pattern discrimination, and clearance of memory traces, regulating cognitive and affective behaviors as well as neural plasticity.
Neuroplasticity is defined as the brain's ability to reorganize itself by forming new neural connections throughout the life, allowing to adapt the brain in response to situations or requests derived from the environment and/or also in response to an internal damage [ 7, 8].
This project combines the field of motor control, which seeks to determine the behavioral consequences of learning to move (e.g., how does the brain adapt the next movement following an error?), and neurophysiology, which attempts to identify the correlates of this motor learning in the firing of neurons in the brain.
A recent elegant study by Neman and colleagues [ 15] investigated whether human BCBMs develop from primed cells in the primary tumor, or if surviving metastatic cells adapt to the brain's unique microenvironment.
Therefore, we adapted the adult brain automated segmentation program of Sajja et al. [21] for use in newborns.
We have also demonstrated that the system can be readily adapted to the brain microvasculature, allowing in principle, localized and inducible modulation of any distinct region of the brain inoculated with the virus.
Because the type III prion population was swa incompetent before inoculation, we assume that it either contained swa-resistant mutants at a level undetectable by our method of competence determination or that mutability increased as the prions adapted to the brain.
The biological mechanisms associated with these proteins and breast cancer brain metastases remain hypothetical, but breast cancer cells that express CD133 or nestin might share features of neural stem cells and might thus be particularly well adapted to the brain microenvironment to initiate brain metastases.
Another explanation for this prolonged PbtO2 decrease may be neurovascular coupling, a mechanism of the brain adapting the CBF to the cerebral ener-gy demand [ 27].
Similarly, when an animal breaks a leg, its brain adapts the gait of the remaining legs so the animal can keep moving.
