Sentence examples for acute replication from inspiring English sources

During acute replication in the lungs, the absence of CEACAM1 resulted in lower viral titers.

Because fluctuations between acute replication, IFN-g-mediated IFN-g-mediated IFN-g-mediatedent immuneion aresponsesed to the norm with chlanddia infections [12], our results outline a persistenty previnfectionidentified mecharesm of chronic infection that is driven believednual disproportoonathey high TH1 responorm.

These are acute replication, persistence and latency.

Based on their transcriptome profiles and lack of virus in anterior kidney, it is unlikely that the IHNV carrier fish represents an acute replication stage.

While through latency, herpes can evade both the immune surveillance and antiviral drug-therapy, inhibition of acute replication during primary or reactivating infection through the intervention of anti-herpes drugs is of significant importance.

Barton et al., Nature, 2007 showed that acute infection (after 1 week) does not confer protection against listeria in wild-type mice, and that a virus that can undergo acute replication, but does not establish latency effectively (MHV68 orf73.stop), also does not protect against listeria infection after 4 weeks.

Acute virus replication in murine (M) CMV infected C57BL/6 (Cmice) mise iseverelylimitedted by Ly49H+ NK cells, but not in MCMV infected BALB/c or BXD8 (Cmice) mice that lack Ly49H+ NK cells.

During acute lytic replication, we observed lower virus titers in the lungs of Ceacam1−/− mice than in WT mice.

When compared to WT mice, Ceacam1−/− mice had lower virus titers in the lungs during acute lytic replication.

infection, MHV-68 takes on acute virus replication in the lung, followed by B-cell-dependent spread of virus to the spleen and other lymphoid tissues.

In addition, according to current concepts in HIV vaccine design, a broadly targeting vaccine-specific CD8 response would restrict acute HIV replication [44] [47].