Died of Acute Interstitial Pneumonia at Our Lady of Lourdes Hospital in Camden, New Jersey.
Acute interstitial syndrome.
Chemotherapeutic agents first cause acute interstitial pancreatitis, which may further progress to acute necrotising pancreatitis.
Main clinical and radiological manifestations of renal sarcoidosis are nephrolithiasis, nephrocalcinosis, and acute interstitial nephritis.
Acute interstitial nephritis (AIN) is an important cause of acute kidney injury, especially in hospitalized patients.
On CT, acute interstitial pancreatitis may be characterised by fluid collections, peripancreatic fat stranding, focal areas of decreased attenuation, or diffuse oedema within the pancreas (Fig. 6) [34].
On urography, acute interstitial nephritis often reveals enlarged kidneys as well as an early, dense, persistent nephrogram, which is similar to the findings of ATN.
On US imaging, acute interstitial nephritis may not be apparent or may present with enlargement of the kidneys as well [9].
Main clinical manifestations are nephrolithiasis, nephrocalcinosis, nephrogenic diabetes insipidus, renal insufficiency, and acute interstitial nephritis with or without granuloma; however, many patients with renal involvement are asymptomatic.
To compare the presenting features and outcome of patients with acute interstitial pneumonia (AIP) with other patients with diffuse alveolar damage (DAD) and with historical control subjects.
It may also be classified by the area of lung affected: lobar pneumonia, bronchial pneumonia and acute interstitial pneumonia; or by the causative organism.
