The phrase "acute brain trauma" is correct and usable in written English.
It can be used in medical contexts to describe a severe injury to the brain that occurs suddenly, often due to an accident or impact.
Example: "The patient was admitted to the hospital with acute brain trauma after the car accident."
Alternatives: "severe brain injury" or "critical head injury."
Staff at the US embassy complained of inexplicable headaches and hearing strange sounds, and it was later discovered that they suffered acute brain trauma.
An injurious immune response may be a common denominator across many neurological disorders, both acute (brain trauma or stroke) and chronic (epilepsy, Parkinson's disease, Alzheimer's for eg.).
In particular, for neurologic injuries, APC reduces damage caused by ischemia/reperfusion in the brain, by acute brain trauma, and by chronic neurodegenerative conditions.
Beneficial therapeutic effects of exogenously administered whole molecule EPO have been described in several animal models of neurologic injury, including occlusive cerebral vascular disease, acute brain trauma, epilepsy, as well as in both autoimmune arthritis and EAE [14], [45], [46].
In patients with acute brain trauma we observed two syndromes causing water-electrolyte disturbance - CDI and CSWS.
In vivo, cannabinoids decrease hippocampal neuronal loss and the volume of brain damage after cerebral ischaemia (Nagayama et al., 1999) and acute brain trauma (Panikashvili et al., 2006).
Acute brain injury from trauma is an important public health problem worldwide.
In this context, β-blockade exposure to modulate the effects of the catecholaminergic storm activated by acute brain injury after trauma or subarachnoid hemorrhage could be beneficial.
This single-center, prospective, observational, 7-month study was based on a cohort of 99 non-infected adult patients admitted to ICUs for elective cardiac surgery, trauma, acute brain injury, and post-operative prolonged ventilation and followed up during ICU stay.
Patients and methods It is a retrospective descriptive study including sixteen patients with acute brain injury (subarachnoid hemorrhage, severe head trauma, and hemorrhagic stroke) and continuous monitoring of ICP who developed a severe ARDS during ICU stay from January 2012 to December 2015 and for which PP was performed.
The oxidative stress is an important factor contributing to delayed neuronal death after acute brain injury by cerebral ischemia or brain trauma, and in neurodegenerative diseases, such as Parkinson's disease and Alzheimer's disease.
