The phrase "activity can induce a" is correct and usable in written English.
It can be used when discussing the effects or outcomes of a specific activity on a particular condition or state.
Example: "Regular physical activity can induce a state of improved mental clarity and focus."
Alternatives: "activity can trigger a" or "activity can lead to a".
In breast cancer cells, up- or downregulation of Notch activity can induce a glycolytic switch.
Such activity can induce a state of reduced sensory processing, similar to the behavioral quiescence induced by low-frequency central thalamus stimulation.
In addition, during less physiological (2), too intense activity at high frequency, MNTs activity can induce a pH drop in skeletal muscle environment to values as low as 6.5 (27), contributing to muscle fatigue (20).
Taken together, these data indicate that selectively blocking CDK4/6 activity can induce a maximum level of G0/G1 enrichment and growth arrest within 24 h without causing cell death with the same dosage for all recombinant CHO cells tested.
As a result, greater effort is required to fulfil the activities of daily living, and performance of these activities can induce an abnormally high level of fatigue.
These studies all demonstrate that abnormal inhibition of endogenous VEGF-A activity can induce maternal pre-eclampsia-like symptoms.
Bursts of synaptic activity can induce long lasting strengthening of excitatory synapses, a phenomenon termed 'long-term potentiation' (LTP), which was discovered in the hippocampus [ 7] and has been observed at virtually every glutamatergic synapse in the brain [ 6].
As NRF2, a regulator of antioxidant activity, can induce HO-1 expression, we next determined whether HO-1 induction via FAC occurred in an NRF2-dependent manner via knockdown of NRF2 in FAC-treated T80 cells.
Such activity can induce platelet, erythrocyte and white cell activation, protein redistribution and accelerated cell aggregation [50].
Furthermore, since aberrant Notch4 activity can induce mammary gland carcinoma in the absence of RBPjκ, a better understanding of the components of RBPjκ-independent oncogenic Notch signaling pathways and their contribution to Notch-induced tumorigenesis would facilitate the deployment of Notch inhibition strategies for effective treatment of breast cancer.
Similarly, hyper-physiological levels of p53 activity can induce pro-oxidant and apoptosis-promoting genes (e.g., NQO1, POX, BAX, PUMA, p66shc), and/or cause an imbalance in expression of antioxidant genes (e.g., MnSOD, PIG12, ALDH4, GPX), and again cause increased oxidative stress [ 38].
