Sentence examples for active survival from inspiring English sources

Cells which survive chemotherapy may have a distinctive biology (e.g. active survival pathways, quiescent cell cycle status) compared to the bulk of cells in the clone, and/or may be protected by the bone marrow niche microenvironment in which they reside.

Within bioaugmentation, critical environmental factors are both predation by protists, and competition by the autochthonous microorganisms, and risk to jeopardize the active survival of the introduced exogenous degraders and consequently negatively affect bioremediation [10].

MECM is a simplified method that is based on the original environmental contour method (ECM) but with modification to be better suited for offshore wind turbines or other systems in which active survival strategies are applied to reduce the system responses in extreme conditions.

These alterations lead to constitutively active survival signaling that enhances the insensitivity of tumour cells to apoptosis induction.

Thus, as a hallmark of HCC, cell proliferation is the result of rebalancing between active apoptosis by DNA damage and active survival by DNA damage repair.

Drug resistance in pancreatic cancer cells is thought to occur mainly as a result of active survival mechanisms and/or inefficient drug delivery because of the fibrotic nature of pancreatic tumours (Olive et al, 2009; Pei et al, 2009).

The combination of gemcitabine and docetaxel, both administered weekly, is well tolerated by elderly and/or poor PS patients, is active (median survival 7 months, 1-year survival 29%) and merits further study.

Apart from active protection, survival time may depend on the tolerance or ability to endure high viral loads, and comparison of the hepatic responses tended to support this possibility.

The ability of these treated neurons to extend neurites acts as an important indication that they remain functionally active following survival of PDT treatment in terms of their regenerative capacity.

Our phospho-proteomic-based pathway activation mapping analysis suggested that the intrinsic resistance of GSCs to cytotoxic drugs is dependent upon constitutively active pro-survival pathways and confirms previous findings reported by other investigators.

Within the active treatment group, survival analysis 36 months from baseline showed that the memantine responders, based on CGIC, had higher rates of survival compared with the non-responders (log rank x²=6.595, p=0.010).