The phrase "active sensitization" is correct and usable in written English.
It can be used in contexts related to psychology, immunology, or any field discussing heightened awareness or responsiveness to stimuli.
Example: "The study focused on the effects of active sensitization in patients with allergies, highlighting how their immune systems reacted to specific allergens."
Alternatives: "heightened sensitization" or "increased sensitivity".
The results suggest that active sensitization per se increases cough reflex sensitivity accompanied by increased inflammatory cells and substance P level in BALF, and antigen challenge further increases them, while simple IgE- and/or IgG-mediated allergic reaction per se or the low intensity of eosinophil infiltration in the airway itself may not affect cough reflex sensitivity in guinea pigs.
The present study confirmed other researchers' investigation that active sensitization per se induces airway eosinophilic inflammation and increase in cough reflex sensitivity [ 11] and our previous data [ 5] that an aerosolized antigen challenge further enhances the airway responses in actively sensitized animals.
Autoimmune disease in humans after active sensitization with brain tissue is a rare event.
The third lesson is around active sensitization and engagement of key players through a well-organized leadership.
We show here that the vast majority of putative disease specific pathological alterations of MS are well reproduced in a patient with brain autoimmune disease induced by active sensitization with brain cells.
Autoimmune disease of the nervous system has occasionally been observed in humans after active sensitization with brain tissue or brain cells, giving rise to acute demyelinating polyradiculoneuritis, acute disseminated encephalomyelitis and in rare cases reflecting an inflammatory demyelinating condition similar to acute multiple sclerosis.
The presence of active TrPs indicates sensitization of muscle nociceptors in both TMD and FMS since high levels of neuroactive mediators [12, 13] and lower pressure pain thresholds [34 37] have been found in active TrPs.
The presence of active TrPs indicate sensitization of peripheral muscular nociceptors since high levels of chemical mediators [33, 34] and greater mechanical hypersensitivity [35, 36] have been found in active TrPs.
The same difference between active and passive sensitization of animals is investigated concerning the antigen-induced late asthmatic response (LAR): LAR develops in actively, but not passively, sensitized guinea pigs [ 25].
The grain boundary activation (DOS, degree of sensitization), the active current density (Ia) and reactivation current density (Ir) were significantly decreased by the nitrogen ion implantation in H2SO4+KSCN solution.
Latent MTrPs can reportedly be activated by a lesion at other sites or within the muscle itself via central sensitization, whereas active MTrPs can be permanently inactivated completely only if the underlying etiologies have been eliminated [ 1- 8].
