The phrase "active nitric" is correct and usable in written English.
It can be used in scientific or technical contexts, particularly in chemistry, to refer to nitric acid in its reactive form.
Example: "The reaction requires active nitric to initiate the process effectively."
Alternatives: "reactive nitric" or "nitric acid in active form".
We hypothesized that adipokine leptin worsens sinusoidal injury by decreasing functionally active nitric oxide synthase 3 (NOS)3 via miR21.
The team found that the blood of Tibetans had more than 10 times the concentration of biologically active nitric oxide breakdown products, such as nitrate and nitrite, than did blood from people who lived at sea level, signifying a high level of nitric oxide in their bloodstreams.
S-nitroso-L-glutathione was found to possess radiosensitising activity, consistent with a possible role of natural thiols in the storing of radiobiologically active nitric oxide adducts derived from sodium nitroprusside.
Vanadia/alumina and molybdenum/alumina catalysts are active for nitric oxide reduction with methanol and dimethyl ether in the presence of excess oxygen in the temperature range 250 500°C.
Glyceryl trinitrate (GTN) (nitroglycerin) and isosorbide-5-mononitrate (5-ISMN) have similar mechanisms of actions and exert their effects through their active metabolite nitric oxide (NO) [1] which is a potent regulator of vascular tone [2].
Based on computer modeling of the active site of nitric oxide synthases (NOS), a series of 10 amidine compounds (9 18) was designed including potential inhibitors that involve the coordination of side-chain functional groups with the iron of the heme cofactor.
Ian M. Wasser, Constantinus F. Martens, Claudio N. Verani, Eva Rentschler, Hong-wei Huang, Pierre Möenne-Loccoz Möenne-Loccozrov, ArnoLevL. RheiN.old and Kenneth D. Karlin, "Synthesis and spectroscopy of m-oxo bridged heme/non-heme diiron complexes: Models for the active site of nitric oxide reductase". Inorg.
Despite the effects of APOSEC on expression and activation of nitric oxide synthases, some immediately occurring effects might (also) be caused directly by biologically active compounds residing in APOSEC.
Anti-vascular endothelial growth factor (anti-VEGF) therapy has been reported to reduce vascular permeability by altering the balance of vaso-active cytokines like nitric oxide and endothelin-1 [ 4] or by directly altering endothelial tight junction proteins [ 5].
NF-κB binds to DNA to enable the initiation of the transcription process of a number of genes, two of them encoding redox-active enzymes: inducible nitric oxide (·NO) synthase (iNOS) and cyclooxygenase-2 (COX-2; which generates superoxide (·O2-) as a by-product) [ 3].
(iii) Glutamate binding to glutamate receptor, ionotropic, N-methyl D-aspartate (GRIN) receptors induces calcium influx inducing CaMKII autophosphorylation. (iv) Active pCaMKII phophorylates neuronal nitric oxide synthase (nNOS), which generates nitric oxide (NO), producing differential effects according to time of day.
