The essence of active GE correlation is choice: Individuals select, modify and create experiences that are correlated with their genetic propensities.
Although there is much to learn about GE interaction (Petrill et al. 2013), I suggest that active GE correlation will ultimately be more enlightening about the developmental interplay in which genotypes use the environment from cells to society to develop into phenotypes.
The Au Pd particles are shown to be more active and correlations made between the catalytic activity and particle radius, surface area and concentration give insight into the mechanism of the catalytic process.
Indeed, it happened accidentally that when there is an important similarity between the actives, there is also an important number of hydrogen bond donors in these actives; the correlation between Sim2Dact-act and HBDactives is 0.36 with p-value = 10−4.
This finding is in contrast with the idea that people modify and select their own environments, which results in (re)active gene environment correlations [ 37, 38].
These were the first MEA recordings of spontaneous activity in the β2 KO mouse, and showed that although individual RGCs were spontaneously active, the correlations in firing of neighbouring RGCs were strongly reduced.
Computational models of cortical networks typically derive irregular spiking by establishing a balance in fast E/I (Shadlen and Newsome, 1998; Renart et al., 2010), which allows inhibitory and excitatory currents to track each other closely in time, resulting in an active de-correlation in spiking (Renart et al., 2010).
Although, H3K9ac and H3K14ac, a hallmark of gene activation exhibit remarkable correlation over active and bivalent promoters as well as distal regulatory elements, a subset of inactive promoters is selectively enriched for H3K14ac.
Clinical studies have shown that TNF protein and mRNA levels are elevated in serum, intestinal tissue, stool of active IBD, in correlation with disease activity [ 4, 7– 9].
An investigation of the electrochemical oxidation of α-Mo2Cand the catalyst's HER activity show a direct correlation between active sites for HER and active sites for catalyst oxidation.
