Sentence examples for activation therapy from inspiring English sources

Exact(32)

Although native (or dietary) vitamin D supplementation can replenish 25-D levels in CKD, there is also a need for VDR activation therapy (calcitriol or selective VDR activators) to correct abnormally low levels of 1,25-D and to control serum PTH levels [ 16, 17].

Some preliminary evidence is provided by one study of depressed adults [ 44], which demonstrated that behavioural activation therapy (which encouraged patients to be more active and not ruminate) was more effective at treating depression than more traditional CBT (with more of an emphasis on cognitive restructuring).

Chemotherapeutic suicide gene therapy approaches are known as gene-directed enzyme prodrug therapy or gene-prodrug activation therapy.

Emerging therapeutic approaches include cervical vagal stimulation, transcutaneous auricular vagal stimulation, baroreceptor activation therapy spinal cord stimulation, ganglionated plexi ablation, renal sympathetic denervation, and left cardiac sympathetic denervation.

Baroreflex activation therapy (BAT) produced by stimulating the carotid sinuses using an implanted device (Rheos) is being studied for the treatment of hypertension, the primary comorbidity of HFpEF.

Initial efficacy is presented of a school-based transdiagnostic group behavioral activation therapy (GBAT) that emphasizes anti-avoidance in vivo exposure.

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Besides these experimental data, observational studies in hemodialysis patients reported improved cardiovascular and all-cause survival among those receiving selective VDR activation therapies.

An earlier screen of different bacteria for nitroreductases suitable for prodrug activation therapies identified YwrO from Bacillus amyloliquefaciens, which selectively reduces the 4-NO2 grofp of CB1954 and was reported to have a modest kinetic advantage relative to E. coli NfsB (Anlezark et al, 2002).

Genetic prodrug-activation therapy is designed to selectively express a non-mammalian metabolic 'suicide' gene in tumour cells such that a coadministered non-toxic prodrug will be converted into its toxic metabolite only within the tumour cells.

Prodrug activation gene therapy for cancer involves expressing prodrug-activating enzymes in tumour cells, so they can be selectively killed by systemically administered prodrug.

We conclude that NfsA should be considered as an alternative prodrug-activating enzyme to NfsB for use in prodrug activation gene therapy or cell/tissue ablation studies using CB1954, nitrofuran antibiotics or DNBM prodrugs.

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