The phrase "activation stress" is correct and usable in written English.
It can be used in contexts related to psychology, physiology, or any field discussing stress responses during activation or engagement in tasks.
Example: "The study focused on the effects of activation stress on cognitive performance during high-pressure situations."
Alternatives: "engagement stress" or "activation pressure".
Additionally, GSK3β siRNA remarkably inhibited Wnt5a-induced RhoA activation, stress fiber formation and cell migration.
For convenience, the randomness in the activation stress is modeled by assigning a random Schmid factor to the dislocations.
This modification consists in the introduction of an equivalent activation stress in the process controlling the plasticity kinetics.
Such gradient pseudoelastic behavior provides an activation stress of 130 MPa for progressive motion of NiTi apparatuses, especially in control and actuation applications.
We find that the activation stress for network breakage and formation of the material controls the gross features of the response of the model, and exhibits a "thermal Bauschinger effect".
Analytic formulas linking yield stress to microscopic interface strength, dislocation source activation stress and other easily obtainable parameters (the Burgers vector, the elastic constants of constituent materials, crystal structure and layer thickness) are provided for all length scales.
Considering that liver fibrogenesis is always accompanied with TGFβ over-activation, stress, HSCs excessive proliferation and severe inflammatory response [1], [6], we thus assume that PP2Cα might be also connected with liver fibrogenesis for its negative role in TGFβ, stress, cell cycle and inflammatory signaling pathways [12] [16].
We therefore investigated whether ATF2 could regulate the activation of stress activated kinases in motoneurons.
Further evidence indicates that Rac activation by stress depends on the magnitudes of the applied stress and cytoskeletal integrity.
Through AMPK activation, metabolic stress represses HSF1, rendering cells vulnerable to proteotoxic stress.
Activation of stress-activated proteins such as JNK is known to enhance TRAIL-induced apoptosis.
