The phrase "activation of a receptor" is correct and usable in written English.
It can be used in contexts related to biology, pharmacology, or biochemistry when discussing how a receptor responds to a stimulus or ligand.
Example: "The activation of a receptor by a neurotransmitter is crucial for signal transmission in the nervous system."
Alternatives: "receptor activation" or "triggering of a receptor".
Some receptors have intrinsic tyrosine kinase activity and phosphorylation sites on the cytoplasmic side of the receptor chains, others recruit tyrosine kinases when activated and, in other cases, activation of a receptor and signalling requires the recruitment of a co-receptor.
Our data indicated that binding to the cell membrane was not enough to provoke a response, involving an intracellular cascade activation of a receptor coupled protein G that activates adenylate cyclase and PKA activities as was proposed in Zhang et al., 2006 [ 52].
The activation of a receptor is driven by the capture of an external ligand[3] and starts with a conformational change followed by a biological chain of events finally detected by the brain.
In this study, we sought to determine whether retroviral insertional mutagenesis could be used to isolate cell mutants, in which the transcriptional activation of a receptor gene has occurred.
We suggest that these genes are regulated by the presence or absence of a particular receptor, rather than by activation of a receptor through growth factor ligation.
This effect is most parsimoniously explained by the additional activation of a receptor "reserve" [46], [47], although the major effect of benzodiazepines may be due to the prolongation of postsynaptic responses [48].
Neurons committed to apoptosis may do so by either activation of a receptor-mediated pathway employing caspase-8 or through an alternative mitochondrial pathway involving oxidative stress.
Following activation of a death receptor, a receptor-specific complex cascade of intracellular events results in apoptosis.
The earlier work assumed activity was associated with activation of a single receptor.
TGF-β signals via activation of a heterooligomeric receptor complex consisting of the type II receptor, TGFβ-RII and the type I receptor ALK5.
In contrast, an increase in receptor binding may be due to an upregulation or activation of a possible receptor reserve (Fan et al., 2003).
