The phrase "activation extent" is correct and usable in written English.
It can be used in contexts related to measuring or describing the degree or range of activation in a particular system or process.
Example: "The activation extent of the enzyme was measured to determine its efficiency under various conditions."
Alternatives: "level of activation" or "degree of activation".
Performance is evaluated primarily in terms of sensitivity (i.e., activation extent, activation magnitude, percent detected trials and effect size estimates) using five different tasks expected to evoke neuronal activity in a distributed set of regions.
Whereas medication appeared to increase activation extent, Figure 2 shows that the spatial extent of activation typically remained lower relative to the control group (see Figures S1 and S2 for activation volumes in the whole brain, and Tables S1 and S2 for details about activation foci).
To gain further insights into the chromatin modifications imposed by gene activation, extent of nucleosome histone acetylation around the map2 promoter region was investigated.
The activation extent of various signal transduction pathways involved in chemo-sensitivity such as the NF-κB pathway, explains how resistant or susceptible a cancer type is towards drugs [ 14].
This suggests that the observed increase in overlap should be largely attributed to enlarged activation extents, that is, spread of activation.
The apparent disparity between absolute levels of integrin activation and extent of aggregation highlights the large level in integrin reserve believed to exist in platelets.
Little is known about the activation and extent of MAPK signaling in the synovium of patients with psoriatic arthritis.
However, Fos expression in the infralimbic cortex was positively correlated with reinstatement behaviour in the current study, indicating that this region is indeed activated and the extent of activation is related to the extent of the reinstatement of the conditioned approach to the CS.
This increase appears to be partially dependent on MEKK1 activation, but the extent of MEKK1 dependence seems to vary depending on the stimuli.
We found that R50E induced transient ERK1/2 activation to the extent similar to that of WT FGF1 (until 3 h after stimulation), but could not maintain high ERK1/2 levels after 6 h in NIH 3T3 cells (Fig. 3A and 3C).
Interestingly, a recent simulation study does suggest that the intensity of the inflammatory response does depend on the phenomenon of pathogenic growth [42], in line with our threshold-based dependence of inflammatory activation on the extent of pathogenic proliferation.
