Sentence examples for activation and deletion from inspiring English sources
Heidstra, R., Welch, D. & Scheres, B. Mosaic analyses using marked activation and deletion clones dissect Arabidopsis SCARECROW action in asymmetric cell division.
Our study demonstrates that both activation and deletion of the CB2 receptor do not relevantly modulate atherogenesis in mice.
In our model, although administration of donor splenocytes alive or apoptotic triggered initial expansion of indirect CD4 T cells, only apoptotic cell infusion was associated with deficient activation and deletion of indirect T cells.
The capacity of bacterial Sags to induce the activation and deletion of T cells expressing T cell receptors (TCR) with a specific subset of TCR β-chain variable (Vβ) regions in mice has been extensively studied [2] [3].
Overall, due to disruption of the neocortical structure as a result of Hh pathway activation and deletion of Rbpj in the Ptc1Lox/Lox NestinCre, RbpjLox/Lox NestinCre and Ptc1Lox/Lox RbpjLox/Lox NestinCre neocortex, the layer-specific expression of Nestin, Sox2 and TuJ1 was perturbed.
In a mouse model of non-small-cell lung cancer (NSCLC) Atg7 deletion did not affect tumor formation caused by kRas activation and p53 deletion.
Here, we have established novel mouse models of neoplasms by liver-specific and biliary-pancreatic Kras activation and PTEN deletion.
We next investigated the potential effects of MYC activation and Rb deletion on the DNA content of hepatocytes in pre-neoplastic livers.
The possibility to control MYC activation and Rb deletion with two different methods (removal of doxycycline and Cre-mediated recombination, respectively) also allowed us to test the possibility that the order of the mutations in this system may influence tumor development.
Indeed, Kras activation and Trp53 deletion can be temporally separated in the pancreas using a combination of the Cre- lox and Flp- FRT systems.
Combined, these results indicate that spontaneous calcium oscillations recorded from GCPs are exclusively mediated by P2Y1R activation and that deletion of these receptors is not accompanied by compensatory mechanisms involving the functional expression of other P2 receptors or alteration in the expression/distribution of the 9acGD3.
