The phrase "activating further" is correct and usable in written English.
It can be used in contexts where you are discussing the process of initiating or enhancing an action or function.
Example: "The software update is designed for activating further features that improve user experience."
Alternatives: "triggering additional" or "initiating further".
Many of the streptococci within the pyogenic cluster express one of a variety of plasminogen activators that associate with the respective host's plasminogen to form an activation complex capable of recruiting and activating further substrate plasminogen to plasmin.
Activated neutrophils also release a plethora of proinflammatory mediators such as IL-8, IP-10 (CXCL-10) [2] and leukotriene B4 [3], recruiting and activating further inflammatory cells, thus enhancing the inflammatory response.
MKK3 appeared to have a relatively high basal activity but was activated further by ischaemia and remained active throughout the time course (Fig. 3A).
Mutation of both Site-1 and Site-2, as reported previously [ 52], resulted in constitutive activation of KCC3A that was not activated further by hypotonic high K+ conditions.
We also found that the activity of WNK1 and WNK3 is markedly elevated and not activated further in response to osmotic stress in cells lacking SPAK/OSR1 activity, suggesting that SPAK/OSR1 can significantly influence WNK isoform activity.
Our data show that LTB4 engagement of BLT1 and BLT2 receptors on osteoclast precursors leads to activation of phospholipase C and calcium release activated channel mediated intracellular calcium flux, which can activate further LTB4 autocrine production.
These MSCs are also able to activate further immunological cascades, including a variety of bone morphogenetic proteins (BMPs), that enhance and activate bone growth in the periphery [13].
The dural afferents might be activated further during dural exposure to inflammatory substances or when the dural mast cells are degranulated [5].
C. Neutrophils attracted to the injured tissues will be activated further by fMLP and IL-1ß via their respective receptors (FPR1, and IL-1R), generate oxidative burst and release proteases, which could be further deleterious to tissues (neutrophil-dependent tissue injury).
Tyrosine phosphorylated IRS1 then recruits downstream effector molecules which in turn activate further downstream signalling pathways.
Over-expression of the membrane signal transducers can activate further down-stream cascades.
